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- PI3K/AKTPI3K/AKT
- PI3K-Akt通路PI3K - Akt signal pathway
- PI3K/Akt/mTORPI3K/Akt/mTOR signal pathway
- PI3k/Akt抑制劑PI3k/Akt inhibitor
- PI3K-AKT信號通路PI3K-AKT signal pathway
- PI3KPI3 K
- p-AKTp-AKT
- 基因Aktgenes Akt
- Wortmannin抑制PI3K途徑對K562細胞增殖和Ara-C誘導的細胞凋亡的影響Effects of PI3K on K562 Cell Proliferation and Apoptosis Induced by Ara-C
- AKT蛋白AKT protein
- Akt通路Akt signal pathway
- 利用信號通路特異性抑制劑抑制實(shí)驗，提示PI3K在A(yíng)DAS細胞向內皮細胞分化中And PI3 kinase inhibitor LY294002 blocked the differentiation of ADAS cells into endothelial cells in vitro.
- PI3K-AktPI3K-Akt
- PI3K／AktPI3K/Akt
- 原癌基因產(chǎn)物c CBL對BCR/ABL酪氨酸激酶活性無(wú)反向調節作用 ,它是連接BCR/ABL與PI3K之間的重要橋梁分子。c CBL Has no regulatory effects on BCR/ABL tyrosine kinase. c CBL is assumed to be the linkage between BCR/ABL and PI3K and subsequently activates PI3K. [
- PKB／AktPKB/Akt
- Akt／PKBAkt kinase/protein kinase B
- Akt／mTORAkt/mTOR
- 作用 2 4h后細胞實(shí)驗組細胞BCL -2蛋白表達較對照組降低 ,說(shuō)明Wortmannin可促進(jìn)K5 62細胞的凋亡。 結論 Wortmannin可以通過(guò)抑制PI3K通路抑制K5 62細胞的增殖。Conclusion Wortmannin could inhibitedtheproliferationofK5 6 2cells ,andwascellcyclespecificagent (CCSA) .
- P13K／AktPI3 K/Akt
