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- FCM檢測K562細胞、雷洛昔芬(2.5mg/L)及CsA( 1mg/L)單獨及聯(lián)合作用于K562/A02細胞一定時(shí)間細胞內DNR濃度。Pretreating K562/A02 cells with raloxifene(2.5mg/L)or CsA( 1mg/L) for 48 hours partially restored the sensitivity of K562/A02 cells to DNR (IC50 were5.98mg/L and 8.15mg/L respectively) but had no effect on K562 cells;
- 采用流式細胞儀(FCM)檢測K562細胞、雷洛昔芬(2.5mg/L)及CsA( 1mg/L)單獨及聯(lián)合作用于K562/A02細胞一定時(shí)間細胞膜上P-gp的表達。and p-glycoprotein expression was detected by fluorometry . Results: The IC50 of DNR for K562/A02 and K562 cells were 23.51mg/L and0.29mg/L respectively.
- K562/A02細胞K562/A02 cell
- 細胞cell
- K562／A02細胞株K562/A02 cell line
- 細胞的cellular
- K562／A02細胞K562/A02 cell
- 經(jīng)肝臟微粒體酶代謝后,FM01、Cur抑制K562細胞株生長(cháng)活性均呈下降趨勢。Both inhibitory activity of FM01 and Cur on K562 cells took decrease tendency after treated with microsomal mixed-function oxidase.
- K562細胞／A02細胞K562 cell/A02 cell
- K562細胞株K562 cell line
- BCR/ABL特異性siRNA真核表達載體構建及其對K562細胞的影響Construction of Eukaryotic Expression Vector of SiRNA Specific for BCR/ABL Fusion Gene and Its Effects on K562 Cells
- 漢防己甲素聯(lián)合屈洛昔芬對K562/A02細胞bcr/abl表達的影響Effect of Tetrandrine in Combination With Droloxifen on the Expression of bcr/abl mRNA and Protein of K562/A02
- 切割bcr/abl核酶的逆轉錄病毒載體構建及其對K562細胞的影響Construction of retrovirus vector of bcr/abl mRNA cleaving ribozyme gene and its effects on K562 cells
- 在BCR/ABL基因組上敲入mRNA不穩定元件可逆轉K562細胞惡性轉化Reversing Malignant Transformation of K562 Cells by Knock in an RNA Destabilizing Element into BCR/ABL Genome
- 轉導野生型p53基因提高K562細胞對紫外線(xiàn)誘導的細胞凋亡的敏感性Enhancement of apoptotic sensitivity induced by UV irradiation on the p53 transducted K562 cells
- Tet、DRL和DNR單獨應用對K562/A02細胞bcr/ablmRNA及蛋白表達均無(wú)影響;The application of single drug of Tet or DRL has no effect on bcr/abl mRNA and BCR/ABL protein expression in K562/A02 cell line.
- As_2S_2與STI 571協(xié)同誘導K562細胞凋亡Synergism of As_2S_2 and STI 571 in inducing apoptosis of K562 cells
- 白藜蘆醇聯(lián)合阿糖胞苷對K562細胞的影響The Inhibition of Resveratrol in Combination with Ara-C on Human Leukemia K562 Cell
- Tet和DRL聯(lián)合應用于48h開(kāi)始下調K562/A02細胞bcr/abl mRNA表達,于72h開(kāi)始下調K562/A02細胞P~(210) BCR/ABL蛋白表達;However,Tet in combination with DRL begins to downregulate bcr/abl mRNA and P~(210) BCR/ABL expression of K562/A02 cells at 48h and 72h respectively.
- 聯(lián)合應用bcr-abl融合基因反義寡核苷酸與c-myb基因反義寡核苷酸對K562細胞作用的研究The Effect of Combining bcr-abl Antisense Phosphorothioate Oligodeoxynucleotides and c-myb Aspo on K562 Cell Line
