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- ObjectiveTo study ultrastructural changes of BGC 823 gastric cancer cells due to the killing effects of the specific cytotoxic T lymphocytes(CTL). 目的研究特異性細胞毒T淋巴細胞（cytotoxicTlymphocyte，CTL）殺傷胃癌細胞BGC?823的超微結構變化。
- Objective To explore the specific cytotoxic T lymphocyte(CTL) response induced by DC pulsed with tumor lysates from multiple myeloma patients. 目的研究負載腫瘤抗原的樹(shù)突細胞能否誘導特異性細胞毒T淋巴細胞反應。
- Another study showed that stimulating the specific cytotoxic lymphocytes maybe the efficient way for chronic hepatitis to clear the virus and gain recovery. 新近研究認識到，通過(guò)刺激特異性CTL應答可能是促進(jìn)慢乙肝患者病毒清除和機體恢復的一種有效途徑。
- Pathological change is chronic and continual advancement,patients can emerge renal inadequacy,uraemia.excitatory autacoid and cytotoxic drug have bad curative effect and major adverse reaction. 病變呈慢性的進(jìn)行性發(fā)展，可逐漸出現腎功能不全、尿毒癥。激素、細胞毒類(lèi)藥物療效欠佳，且有較大的不良反應。
- Dendritic cells (DCs) are the most potent antigen presenting cells (APCs) that prime naive T lymphocytes and active antigen specific cytotoxic T lymphocytes(CTLs), which makes DC a promising tools for immunotherapy of cancer . 樹(shù)突狀細胞(Dendritic Cells,DCs)作為功能最強的抗原提呈細胞，在惡性腫瘤的免疫治療中起著(zhù)重要的作用。
- CLT can induce apoptosis in vitro in human colon cancer cell line CCL 229.The apoptosis inducing effect may be G1 phase specific. 克霉唑對人結腸癌細胞系CCL229有明顯的凋亡誘導作用,并且能特異地誘導G1期細胞凋亡。
- Adriamycin is frequently used in combination chemotherapy regiments with other cytotoxic drugs. 阿霉素常與其他細胞毒藥物合用于化療方案。
- The mechanism by which chronic-phase CML cells become resistant to conventional cytotoxic drugs, such as hydroxyurea, is unknown. 慢性期CML細胞對常規細胞毒藥物，如羥基脲產(chǎn)生抗藥性的機理還不清楚。
- Specific cytotoxic T lymphocy te 特異性細胞毒性T淋巴細胞
- Antibody-drug conjugates enhance the antitumor effects of antibodies and reduce adverse systemic effects of potent cytotoxic drugs. 抗體偶聯(lián)藥物能提高抗體的抗腫瘤能力并減少有效細胞毒性藥物的系統性副作用。
- Specific cytotoxic T lymphocytes 特異性細胞毒性T淋巴細胞
- The most effective cytotoxic drugs for treatment of metastatic breast cancer are capecitabine, doxorubicin, gemcitabine, the taxanes paclitaxel and docetaxel, and vinorelbine. 治療轉移性乳腺癌的最有效細胞毒類(lèi)藥有卡培他濱、阿霉素、二氟脫氧胞嘧啶、紫杉烷類(lèi)（紫杉醇和紫杉萜[泰素帝]）、長(cháng)春瑞濱等。
- Conclusion: In vitro the conjugation showed a highly specific cytotoxicity upon phagocytes. 結論：標記抗體在體外對單核-巨噬細胞顯示了高度特異的殺傷活性。
- Oonolusion The conjugation can show a highly specific cytotoxicity upon mononuclear-macrophage in vitro. 結論：標記抗體在體外對單核、巨噬細胞顯示了相對特異的殺傷活性。
- Objective To investigate the impacts of cytotoxic drugs on APE/ref-1 expression in A549 cell, in order to find the potential relationship between APE/ref-1 and chemoresistance of NSCLC. 目的 研究細胞毒性藥物對A549細胞APE/ref-1表達的影響,初步探討APE/ref-1與NSCLC耐藥性產(chǎn)生的相關(guān)性。
- ATP-TCA was used to detect the sensitivity of 35 specimens of fresh cervical cancer to six cytotoxic drugs as follows: paclitaxel (TAX), cisplatin (DDP), bleomycin (BLM), gemcitabine (GEM), 5-fluoruracil (5-Fu), irinotecan (CPT-11). 采用ATP-TCA法對35例宮頸癌患者的新鮮癌組織進(jìn)行體外藥物敏感性測定，分別檢測對泰素(TAX)、順鉑(DDP)、博來(lái)霉素(BLM)、健擇(GEM)、5-氟尿嘧啶(5-Fu)、依立替康(CPT-11)6種化療藥物的敏感性。
- Methods A549 cells were treated with different cytotoxic drugs for 24hours. The changes of APE/ref-1 expression in A549 after drug-treatment were detected by real time PCR, western blot and streptavidin peroxidase immunohistochemistry staining. 方法 通過(guò)免疫組化染色明確APE/ref-1在A(yíng)549細胞中的表達后,以特定濃度的臨床常用細胞毒性藥物對A549細胞進(jìn)行誘導,采用實(shí)時(shí)熒光定量PCR、Western Blot和免疫組化技術(shù)檢測藥物誘導前后A549細胞內APE/ref-1表達的變化情況。
- tumor-associated phase specific antigen TAPSA
- tumor-associated phase specific antigen(TAPSA) 腫瘤相關(guān)相特異性抗原
- The child is going through a difficult phase. 那孩子正經(jīng)歷困難的階段。
