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- CD11/CD18CD11/CD18
- CD18基因CD18 gene
- CD11b／CD18CD11b/CD18
- CD11a/CD18CD11 a/CD18
- CD11b/CD18CD11b/CD18
- CD11c／CD18CD11c/CD18
- CD18淋巴細胞抗原Lymphocyte antigen CD18
- 缺血預適應阻抑犬冠狀竇血中性粒細胞CD11b/CD18的表達Ischemic preconditioning attenuates the expression of neutrophils CD 11b/CD 18 in coronary sinus blood of dog
- 冠心病人血漿可溶性細胞粘附分子和白細胞CD18表達的動(dòng)態(tài)變化The Diurnal Variation of Plasma Soluble Cell Adhesive Molecule and Leukocyte CD18 Expression in Patients with Coronary Heart Disease
- CD11/CD18介導PMN－EC粘附在燒傷早期PMN損傷血管內皮細胞中的作用Study of the effects of CD11/CD18-mediated PMN-EC adhesion on PMNinduced EC damages in the early stage postburn in vitro
- 燈盞細辛注射液對急性腦梗死患者血清粘附分子sICAM-1和CD11b/CD18的影響Effect of Erigeron Injection on Expresions of Adhesion molecules CD11b/CD18 and Solution Cell Adhesion Molecule-1 in Peripheral Blood of Patients with Acute Cerebral Infarction(ACI)
- 目的：探討燒傷血清作用下中性粒細胞CD11/CD18和內皮細胞ICAM－1表達的變化。Objective: To observe the changes of expression of CD11/CD18 on polymorphonuclear neutrophils (PMNs) and ICAM-1 on endothelial cells (ECs) under the stimulation of burn serum.
- 肺部感染患者外周血中性粒細胞黏附分子CD11b CD18髓過(guò)氧化物酶的變化及臨床意義Changes of adhering molecules CD11b,CD18,myeloperoxidase(MPO) expression of the neutrophil in peripheral blood of pneumonia patients
- ACS患者血清E 選擇素水平、中性粒細胞和單核細胞膜CD11b/CD18的表達增加與斑塊不穩定性有關(guān)The serum levels of soluble E-selectin and expression of CD11b/CD18 on neutrophils and monocytes in patients with ACS are related with unstability of plaques.
- 結果發(fā)現：CD18的表達為PMN快相增加粘附力的主要分子基礎，而ELAM－1、ICAM－1則為VEC慢相增加粘附力的主要分子基礎。It was found that the main molecular basis of the rapid increase of PMN adhesive ability was CDI8 expression and that of the delayed increase of VEC adhesive ability was the expression of either ELAM-1 or ICAM-1.
- 提示：燒傷血清可剌激PMN和EC分別表達CD11a/CD18、CD11b/CD18和ICAM－1，從而加強PMN－EC粘附，在燒傷后PMN－EC粘附中起重要作用The results suggested that burnt serum could enhance adhesive force between PMN and EC, stimulate expressions of CD11a/CD18,CD11b/CD18 and ICAM?1.Therefore,burnt serum played a great role in PMN?EC adhesion after burn injury. CD11a/CD18, CD11b/CD18 and ICAM?1 were the molecular base of PMN?EC adhesion.
- 丹參能降低黏附分子 CD11a/ CD18、CD11b/ CD18表達 ,抑制 PMN EC黏附 ,改善微循環(huán)及減輕 PMN EC黏附所致的組織損傷 ,有助于 AP的早期治療。Salvia miltiorrhiza can lower the CD11a/CD18,CD11b/CD18 expression, inhibit PMNEC adhesion, improve the microcirculation, ease the tissue damage produced by PMNEC adhesion and help the AP treatment in the early period.
- 嚴重燒傷能使外周血PMNCD11b/CD18表達明顯增高，CD11b/CD18不但介導基礎狀態(tài)下的PMN粘附，更是介導燒傷后引起的大量PMN與PMEC粘附的主要分子。Severe burn could induce the expression of PMN CD11b/CD18 which may be the main adhesion molecule participating in the increasing PMN adhesion to EC due to severe burns.
- 方法:采用DNA重組技術(shù)構建真核表達質(zhì)粒pTRE-Tight-CFP-CD11b和pTRE-Tight-YFP-CD18(CD11b和CD18是Mac-1的兩個(gè)亞基),通過(guò)脂質(zhì)體介導共轉染CHO細胞,用熒光顯微鏡觀(guān)察轉染后陽(yáng)性細胞;Methods: The eukaryotic expression vectors pTRE-Tight-CFP-CN11b and pTRE-Tight-YFP-CD18 were constructed by recombinant DNA technique. The 2 vectors were co-transfected into CHO cells using liposome to fuse CD11b and CD18: the 2 subunits of Mac-1. Fluorescence microscope was used to observe the cyan fluorescence and the yellow fluorescence of Mac-1-FP.
