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- 緩激肽B2受體阻斷物bradykinin B2 receptors antagonist
- 緩激肽B2受體bradykinin B2 receptor
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- 緩激肽及其受體與糖尿病腎病Bradykinin, bradykinin receptors and diabetic nephropathy
- 烷不溶物heptane insoluble matter
- 那麼，能阻斷緩激肽受體的拮抗劑，必定可以防止這些受體造成的痛覺(jué)受器活化。Bradykinin powerfully stimulates nociceptors, and an antagonist that blocks its receptors would certainly prevent those receptors from activating nociceptors.
- ATP敏感性鉀通道蛋白在緩激肽選擇性開(kāi)放血腫瘤屏障中的表達The Expression of ATP-Sensitive Potassium Channel Protein in Bradykinin Induced Blood Brain Tumor Barrier Permeability Increase
- 緩激肽體外誘導牙髓組織甲硫氨酸腦啡肽及亮氨酸腦啡肽產(chǎn)生和釋放的實(shí)驗研究In vitro production and release of Met-enkephalin, Leu-enkephalin in tooth pulp induced by bradykinin
- 從而提示：內毒素引起CGRP釋放是通過(guò)炎癥介質(zhì)前列腺素、緩激肽、血小板激活因子和組胺介導的，阻斷或減少炎癥介質(zhì)的產(chǎn)生可望減輕內毒素血癥時(shí)CGRP的過(guò)量釋放。The data suggest that PGs, BK, PAF and histamine are involved in the ETX triggered release of CGRP from perivascular nerves in the rat isolated MAB. Blocking the inflammatory mediators might be beneficial in reducing excess release of CGRP during the endotoxicosis.
