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- 白三烯C4（LTC4）Leukotriene CA
- 白三烯C4Leukotriene C4
- 白三烯C4合成酶Leukotriene C4 synthase
- 結論 :LTC4 能上調 16 HBE及ASMC兩種細胞ET 1mRNA的表達并刺激ET 1釋放 ,這可能是白三烯致哮喘氣道炎癥及重塑的重要機制之一。Conclutions:LTC 4 induces over expression of ET 1 mRNA and ET 1 release in 16 HBE and SMCS,which may be one of the important mechanism of LTs in airway inflammation and reconstraction in asthma.
- 白三烯E4Leukotriene E4
- 尿白三烯FAUrinary leukotriene E4
- 白三烯受體1CysLT1R
- 他們的方法是加進(jìn)額外的碳氫基，強化C4碳鏈的結構，使之不易斷裂。They found a way to stiffen the C4 chains by propping them up with extra hydrocarbon groups.
- 白三烯拮抗劑Leukotriene antagonist
- C4.5C4.5
- 抗白三烯治療Leukotriene modifiers
- C4烴C4 hydrocarbon
- 半胱氨酸白三烯Cysteinyl-containing leukotrienes
- 細菌C4bacterium CA
- 白三烯受體拮抗藥Leukotrienes receptor antagonist
- C4 植物C4 plant
- 白三烯及其受體與哮喘的關(guān)系Leukotriene and its receptor in relation with asthma
- C4烯烴C4 olefins
- 白三烯受體拮抗劑對哮喘患兒血清ECP的影響The effects of leukotrienes receptor antagonist on serum ECP of asthmatic children
- 補體C4complement C4
