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- 乳腺癌細胞Bcap-37breast carcinoma Bcap-37
- 亞硒酸鈉對Bcap-37人乳腺癌細胞體外增殖及3種酶細胞化學(xué)的影響THE EFFECTS OF SODIUM SELENITE ON PROLIFERATION AND 3 KINDS OF ENZYME CYTOCHEMISTRY OF Bcap-37 HUMAN MAMMARY TUMOR CELLS IN VITRO
- CDP抑制乳腺癌細胞T47D生長(cháng)及逆轉p16基因甲基化的作用The Research on the Growth Inhibition of T47D Cell and Reversion of p16 Hypermethylation by CDP
- VES聯(lián)合ADM作用24h后Bcap-37細胞表面Fas表達增強。Cell surface Fas expression increased after being treated with VES combined with ADM.
- 低氧環(huán)境下羅勒多糖對乳腺癌細胞TIMPs mRNA表達的影響Effect of basil polysaccharide on mRNA expression of tissue inhibitor of metalloproteinases in human breast cancer cell line cultured under hypoxia
- 流式細胞儀flow cytometry
- VES作用后乳腺癌細胞Fas蛋白水平和細胞表面Fas表達升高。Fas protein level and cell (surface) Fas expression in cancer cells increased after the administration of VES.
- 流式細胞術(shù)flow cytometry
- 鱗狀細胞癌squamous cell carcinoma
- 乳腺癌細胞breast cancer cell
- 瘤細胞oncocyte
- 而且STT能夠降低BEL-7402細胞的抗凋亡蛋白Bcl-2表達，并能夠降低Bcap-37細胞的細胞周期蛋白cyclin D1和cyclin E水平。In addition, STT has GO/G1 arrest effect on cell cycle of Bcap-37 cells.
- Ｇ細胞G cell
- Ｉ細胞I cell
- Ｋ細胞K cell
- VES聯(lián)合化療藥物作用后乳腺癌細胞表面Fas表達顯著(zhù)增強。Fas expression in cancer cells increased after the co-administration of VES and chemotherapy drugs.
- 乳腺癌細胞株,breast cancer cells
- 生物信息學(xué)分析表明,FHR在對Bcap鄄37細胞作用的過(guò)程中,該32條差異表達基因與Bcap鄄37細胞凋亡有關(guān),其中包括CTNNB1、TSSC3、IGFBP4、IGFBP6、GADD34、TNFRSF10B、Caspase鄄9和PCNA等。Bioinformatic analyses showed that the 32 genes, including CTNNB1 , TSSC3, IGFBP4, IGFBP6, GADD34, TNFRSF10B, Caspase-9, and PCNA, related with ap optosis of Bcap-37 cells when treated with FHR.
- 乳腺癌細胞株human breast cancer cell line
