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- Objective:To study the relation between severe oligospermia, azoospermia and abnormal chromosome karyotypes. 目的:探討男性嚴重少精子癥和無(wú)精子癥與染色體異常的關(guān)系。
- FSH and LH in oligospermia were higher than in normal semen, but the differences were not significant (P>0.05). 少精癥組FSH、LH雖均較正常精液組高，但無(wú)顯著(zhù)性意義（P>0.;05）。
- Conclusion Mutation of Exon A in AR gene plays an important part in infertile men with oligospermia. 結論雄性激素受體基因外顯子A即基因轉錄激活區的突變是造成少精不育的重要原因。
- Objective: To study the meaning of the karyotype analysis in oligospermia and azoospermia. 目的探討染色體核型分析在少精子及無(wú)精子者中存在的意義。
- The patients with oligospermia and asthnospermia, Group A was higher than group B in normal sperm morphology percent. 在少精組及弱精組中A組精子正常形態(tài)率高于B組，差異有統計學(xué)意義。
- Conclusion: chromosomal abnormalities play an important role in oligospermia and azoospermia. 結論染色體異常是導致少、弱精子及無(wú)精子癥的重要因素之一。
- CONCLUSION: Both of abnormal chromosome karyotype and Y-chromosome microdeletion are important to cause azoospermia and severe oligospermia. 結論：染色體核型異常和Y染色體微缺失均與無(wú)精子癥和嚴重少精子癥的發(fā)生有關(guān)。
- Objective To investigate the clinical value of the determinations of serum sex hormone in the patients with oligospermia and azoospermia. 目的探討少精、無(wú)精癥患者血清性激素檢測的臨床應用價(jià)值。
- Objective:To investigate the effects of Shengjingbao on spermatogenesis in the mouse model of oligospermia and its action mechanisms. 目的:研究"生精寶"對小鼠少精子癥模型生精功能的作用并探討其機制。
- Objective: To study the imprinting status of H19 in oligospermia and asthenospermia and to investigate the correlation between H19 and spermatogenesiss disturbance. 目的:通過(guò)研究印跡基因H19在少弱精子癥中的印跡狀態(tài),探討H19與精子發(fā)生障礙的相關(guān)性。
- Objective In order to efficiently evaluate the result to treat oligospermia or asthenozoospermia with combination of Chinese Traditional and Western medicine. 摘要目的采用新標準更科學(xué)地評估中西醫結合治療少弱精子性不育癥的療效。
- Results FSH and LH in azoospermia and severe oligospermia were all significantly higher than in normal semen (P<0.01), and FSH was raised more highly than LH each other. 結果無(wú)精癥組及重度少精癥組FSH、LH均顯著(zhù)高于正常精液組（P<0.;01），且FSH升高幅度均大于LH。
- BACKGRONUD &AIM: To investigate the relationship between spermatogenesis disorder and genetic defects of patients with azoospermia or severe oligospermia. 摘要背景與目的：探討無(wú)精子癥和嚴重少精子癥患者的遺傳缺陷與精子生成障礙的關(guān)系。
- Adverse effects were bilateral nosocomial pneumonia in 2 patients, late endocrine dysfunction in 3 patients, and oligospermia in 9 patients.There were no deaths. 不良效果是2例患者雙側醫院性肺炎，3例患者晚內分泌功能失調，9例患者少精液癥，但是沒(méi)有患者死亡。
- Methods: The periphery blood lymphocyte was cultured and the G-banding chromosome was analyzed on 394 male patients with the severe oligospermia and azoospermia. 方法:對364例嚴重少精子癥和無(wú)精子癥患者進(jìn)行外周血淋巴細胞培養,G顯帶染色體核型分析。
- BACKGRONUD & AIM: To investigate the relationship between spermatogenesis disorder and genetic defects of patients with azoospermia or severe oligospermia. 背景與目的：探討無(wú)精子癥和嚴重少精子癥患者的遺傳缺陷與精子生成障礙的關(guān)系。
- Compared with the normal controls.PCNA proliferation index (PI) was lower (P<0.001) and apoptosis index (AI) higher (P<0.01) in testes of oligospermia patients. 結果與正常睪丸相比，治療前患者PCNA增殖指數(PI)偏低(P<0.;001)，凋亡指數(AI)較高(P<0
- Results: The proportion of autosomal structure abnormalities was 5.1% (23/454) among the patients with idiopathic oligospermia and azoospermia, while that of chromosomal polymorphism was 8.8%(40/454). 結果在454例特發(fā)性生精異?；颊咧杏?3(5.;1%25)例涉及常染色體結構異常;40(8
- Conclusions Tamoxifen significantly decreases the incidence of germ cell apoptosis and improve the spermatogenic function.Tamoxifen is recommended to advocate in cases of idiopathic oligospermia. 結論少精子癥精子減少的主要原因之一是由于生精細胞增殖能力減低及凋亡增加，它莫昔芬可糾正生精細胞增殖與凋亡的失衡狀態(tài)，改善患者生精功能及生育能力。
- It is mainly indicated for male loss of libido, short and small penis, dysfunction of erection and premature ejaculation, no activity of spermatozoa, oligospermia and other conditions. 藏藥與其它藥的區別在于藏藥多為主藥材及金訶為藥引，不加別的成份，而中藥往往以淀粉、紅糖等作為輔料。
