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- The mRNA expressed in human pancreas cells was copied with reverse transcriptase to create a "library" of human DNA. 人胰腺細胞中表達的mRNA用逆轉錄酶進(jìn)行復制并建立了人類(lèi)DNA的文庫。
- The style of K-ras gene point mutation at codon 12 was GAT in human pancreatic cancer cell line. 人胰腺癌細胞株P(guān)ANC-1存在K-ras基因的點(diǎn)突變,其突變方式為CAT。
- The human pancreatic adenocarcinoma cell lines AsPC-1, Bxpc-3, were grown in RPMI-1640 medium containing 10%FCS. 人胰腺癌細胞株 ASPC－lx BXPC－3購自上海細胞所，復蘇后在含 10%25胎牛血清的 RPMI－1640培養基內培養傳代。 用逆轉錄聚合酶鏈反應
- Human pancreatic ribonuclease has 128 amino acid residues. 人的胰腺核糖核酸酶有128個(gè)氨基酸殘基。
- K ras gene point mutation at codon 12 was found in human pancreatic carcinoma cell line PC 2, and the mutation style was CGT. 人胰腺癌細胞株P(guān)C 2存在K ras基因點(diǎn)突變 ,突變方式為CGT。
- Conclusions: there were over expressions of PTCH mRNA and Smo mRNA in human pancreatic cancer tissues and human pancreatic cancer cell lines. 小結:PTCH和Smo mRNA在人胰腺癌細胞系和組織中均存在過(guò)表達;
- Abstract：Objective To observe the effect of photodynamic therapy(PDT)on human pancreatic carcinoma cell lines in vitro. 摘 要：目的 探討光動(dòng)力作用(PDT)對體外培養的人胰腺癌細胞的殺傷效應。
- METHODS:RT-PCR analysis was used to detect the expression level of KiSS-1 mRNA in human pancreatic cancer cell line AsPC-1 and tissues of 40 cases of pancreatic cancer, 27 cases of their metastastic site and 9 cases of normal pancreatic tissues. 方法:應用RT-PCR方法檢測KiSS-1基因mRNA在人胰腺癌細胞株AsPC-1、40例胰腺癌及27例轉移灶和9例正常胰腺組織中的表達情況。
- PACAP6 38 inhibited this effect of PACAP1 38. Conclusions: PACAP1 38 can stimulate the proliferation of human pancreatic cancer cell strain ASPC 1. The growth promoting effect of PACAP1 38 were mediated by the corresponding receptors. 結論:PACAP1-38促進(jìn)ASPC-1的增殖;
- They hitched the genes for these three factors onto a virus that infects another type of pancreatic cell, known as an exocrine cell. 他們把這三組因子的基因搭載到一種病毒上，再用病毒感染被稱(chēng)作外分泌細胞的另一種類(lèi)型的胰腺細胞。
- Methods Human pancreatic carcinoma cell lines SW1990, Capan-1 (Cap), AspC-1 (ASPC), P3, PANC-1 (Pan-1) and MIAPaCa-2 (MIA) were tested for MT level and radio-sensitivity using ELISA and clonogenic assay, respectively. 方法分別采用外源性鋅離子及反義寡聚核酸(AS)轉染P3、Pan-1、MIA、SW1990、Cap、ASPC等6株胰腺癌細胞,調節M(mǎn)T表達,隨后應用ELISA及集落實(shí)驗檢測MT表達水平及放射敏感性指標存活分數(SF2)的變化。
- Last week, Geron Corp. said it had transformed human embryonic stem cells into the pancreatic cells that produce insulin. 他說(shuō)以前努力用人類(lèi)干細胞制造胰島素，制造出來(lái)的量很少，或者存在錯誤。
- Methods Using the quantitative reverse transcription PCR procedure,we observed the expression of nm23 mRNA in human pancreatic cancer JF305 cell sublines with different metastatic potential. 方法 在建立不同轉移潛能的人胰腺癌JF305 單克隆細胞亞系的基礎上，應用定量逆轉錄多聚酶鏈式反應技術(shù)（RT?PCR）檢測不同轉移潛能的人胰腺癌JF305 單克隆細胞亞系細胞中nm23 mRNA 的表達。
- The MTT assay and median-effect principle were used to observe theinteraction of 5-fluorouracil ,cisplatin and epirubicin on human pancreatic carcinoma cell line Aspc-1.Results: l. 采用MTT測定5一FU、DDP和E一ADM單用及兩兩合用時(shí)對細胞株AsPc 一1的抑制作用，并應用中效原理判定藥物合用的效果。
- Using a human pancreatic cancer cell line, she and her team found that adding thymoquinone killed approximately 80 percent of the cancer cells. 使用人體胰腺癌細胞株，她的小組發(fā)現加入百里香醌能夠殺滅80%25的癌細胞。
- PACAP1 38 stimulated the proliferation of human pancreatic cancer cells ASPC 1 which were dose dependent. PACAP1-38促進(jìn)ASPC-1的生長(cháng),促進(jìn)ASPC-1細胞cAMP、Ca2+的生成,且呈劑量依賴(lài)性;
- The result suggests that the energy metabolism, substance digestion, anaerobic glycolysis of liver and pancreatic cell, and aerobic metabolism of pancreatic cell are strengthend after lighting. 提示光照后;肝、胰細胞的能量代謝、物質(zhì)消化、無(wú)氧酵解和胰細胞的有氧代謝均有增強.
- The cell with the most content in alvine pancreas cell implement what be? 腸胰細胞中含量最多的細胞器是什么?
- Objective To investigate the expression of substance P (SP), neurokinin-1 receptor (NK-1R) and neutral endopeptidase (NEP) in human pancreatic cancer. 目的 了解P物質(zhì)(SP)、神經(jīng)激肽-1受體(NK-1R)及中性肽鏈內切酶(NEP)在胰腺癌細胞中表達。
- ObjectiveTo investigate the therapeutic effect and the mechanism of photodynamic therapy (PDT) on human pancreatic cancer xenograft in nude mice. 目的研究光動(dòng)力療法 (photodynamictherapy ,PDT)對裸鼠胰腺移植癌的治療效果及血管損傷在其中的作用 ,為臨床應用PDT治療胰腺癌提供依據。
