There are some neutralization locations in the VP3 of hepatitis A virus and VP2 and VP3 of poliovirus indicates that the YP3 of AEV maybe in some degree function as protection protein resisting infection of virus.
英
美
- 小RNA病毒科中,結構蛋白VP3被認為與病毒的持續感染、T細胞免疫以及與病毒受體的相互作用有關(guān),近期研究還表明AEV的結構蛋白VP3能夠誘導宿主細胞的凋亡。
