The pathogenesy of smoke inhalation acute lung injury/acute respiratory distress syndrome(ALI/ARDS) is not completely clear,perhaps concerned with oxidative damage,the change of pulmonary surfactant and cytokine,and apoptosis.
英
美
- 目前煙霧吸入性急性肺損傷(ALI)/急性呼吸窘迫綜合征(ARDS)的發(fā)病機制尚未完全明確,可能與煙霧所致的氧化性損傷、肺表面活性物質(zhì)變化、細胞因子改變、細胞凋亡的發(fā)生有關(guān)。
