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- F-actinF-actin
- β-actinβ-actin
- actin基因actin gene
- α-SM-actinα - smooth muscle - actin
- actin結合活性actin binding activity
- β-actin基因β-actin gene
- -βactin啟動(dòng)子β-actin promoter
- 雞β-actin啟動(dòng)子Chicken β -actin promoter
- EMA、Actin、S-100、ER陰性;Negatived for EMA,Actin,S-100,ER;
- 標記F-actin,熒光顯微鏡下觀(guān)察。F-actin was labled and examined using fluorescence microscope.
- 高糖組F-actin熒光強度為對照組的44.5%;High glucose can decrease the fluorescent intensity of F-actm to 44.5%25 (p<0.01);
- MOMA-2表達增加,SM-actin表達降低(P<0.05)。Even,upregulation of MOMA-2 and downregulation of SM-actin were also detected in latter(P<0.05).
- 電離輻射對血管內皮細胞骨架蛋白F-actin影響的激光共聚焦顯微鏡觀(guān)察The Observation of the Influence of Ionizing Radiation on the Image of F-actin in Vascular Endothelial Cells with Confocal Laser Scanning Microscope
- 結果表明,LIM結構域2在hhLIM與actin相互作用及調節actin細胞骨架組構中起決定性作用.In conclusion, LIM domain 2 at the C-terminus of hhLIM plays a central role in F-actin polymerization and cytoskeleton stabilization, whereas the first LIM domain is essential for the nuclear localization of hhLIM.
- 其ANA以核膜型為主 (7 14 ) ; AIH患者ANA抗體未見(jiàn)特定的熒光類(lèi)型 ,而抗 Actin僅見(jiàn)于A(yíng)IH者。No specific ANA pattern were seen in AIH by IIF, and anti Actin was present only in patients with AIH.
- 4Hz、12Hz和20Hz的100dB的次聲作用30min/d后,可誘導F-actin表達的增強,這種改變在8h后仍未見(jiàn)減弱。F-actin expression can be increased in osteoblast MC3T3 exposed 30min/d by low intensity infrasound with output intensity of 100dB and frequency of 4Hz,12Hz,20Hz. The expression increasing of F-actin still can be founded at the 8th hour after exposed under the low intensity infrasound.
- 通過(guò)PCR技術(shù) ,我們從文庫中釣取到EGP、2 3kD膜蛋白、Actin和GCP的cDNA ,其中GCP基因為低豐度表達基因。By using PCR we obtained the cDNAs of EGP,23KD membrane protein,Actin and GCP from our cDNA libraries,GCP is a low|abundant expressed gene.
- 和a一SMA(a一smooth musele actin)的表達。 5反轉錄聚合酶鏈反應(reverse transcription一polymerase ehain reaction,mRNA expressions of TGF-B1 and PPAR r were detected by Reverse transcription-polymerase chain reaction (RT-RCR).
- 免疫組織化學(xué)染色結果顯示:Vimentin、Actin標記細胞呈陽(yáng)性反應。 Desmin、AACT、EMA、Cytokeratin和S-100標記為陰性。The results revealed that the cells were positive for vimentin, actin and negative for desmin, AACT, EMA, cytokeratin and S-100protein.
- 免疫組化結果:瘤細胞desmin及vimentin呈彌漫強陽(yáng)性表達,actin、CD34、ER和PR染色稍弱、呈灶狀分布,而S-100蛋白、NF和CK均陰性。Immunohistochemically, the tumor cells were strong positive for vimentin and desmin, only local positive for CD34, ER and PR, but all tumor cells were negative for S-100 protein, NF and CK.