SDS-PAGE and IEF analyses showed that the mutants without PI6.82 polypeptide in common while forming / increasing other polypeptide indicating that the mutants were controlled by structural genes.

 
  • SDS-PAGE和IEF分析表明這些突變體在共同缺失1條pI6。82多肽的同時(shí),或形成新的多肽,或其他多肽表現量增加,這些突變體是由結構基因控制的。
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