Objectives: We hypothesized that deletion or pharmacologic inhibition of PI3K would impair the lung inflammatory response to the prototypic gram-positive bacterial pathogen Streptococcus pneumoniae.

 
  • 目的:我們假設敲除或者藥物抑制PI3K將損害肺臟對于革蘭氏陽(yáng)性菌肺炎鏈球菌的炎癥反應。
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