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- KGF and KGFR are expressed in Hela cells. Hela細胞中有KGF和KGFR的表達;
- Hela cells were also injected to normal BALB/c and Kunming mice. 同時(shí)用Hela細胞作為陽(yáng)性腫瘤細胞對照，并接種于免疫功能正常的BALB/c小鼠和昆明種小鼠。
- HPV16 E7 was expressed efficiently in HEK-293 and HeLa cells. 熒光顯微鏡下,轉染后HEK-293細胞和HeLa細胞表達綠色熒光蛋白。
- Either the cell growth inhibitory rate or the colony inhibitory rate of the HeLa cells was 100%(P<0.01) after being exposed to 12 Gy irradiation, meanwhile, the value of 2,3-DPG was nearly unchanged(P>0.05). 12 Gy的輻射劑量能夠完全抑制腫瘤細胞的增生(細胞生長(cháng)抑制率及克隆形成抑制率均為100%25;P<0.;01);且不影響紅細胞的攜氧功能(不輻射與輻射后的2;3 DPG值無(wú)顯著(zhù)性差異;P>0
- Methods: (1)To detect toxic effect of PAP to Hela cell by MTT assay. 方法:(1)用MTT法檢測PAP對Hela細胞的毒性作用;
- RT-PCR showed that E1A gene had a stable transfection in Hela cells and obviously down-regulated Her-2 expression. RT-PCR結果顯示:E1A基因在Hela細胞中能穩定表達,E1A基因的轉染明顯降低了Her-2基因在Hela細胞中的表達水平;
- Objective: To study the fate of LAK cell and rescuing effect of Astragalus polysaccharide (APS) during and following assault on Hela cells. 目的：進(jìn)一步探討LAK細胞攻擊Hela細胞后的歸宿及黃芪多糖（APS）的影響。
- Low dose cisplatin can enhance the sensitivity of radiotherapy through change cell cycle of Hela cell lines. 小劑量順鉑可通過(guò)改變培養的人Hela細胞周期 ,誘導細胞G2 /M期延遲從而提高放療敏感性 ,可能與其內在的 p5 3基因表達改變有關(guān)。
- At 12 h, the H_2O_2 level in HeLa cells and AsPC-1 cells were similar to that of the control group. 結論:As2O3誘導HeLa細胞和AsPC-1細胞凋亡與細胞內的H2O2水平改變有關(guān),而與HPV的感染無(wú)明顯相關(guān)性。
- The s-lap/GFP fusion protein can express in Hela cells and locate in entire cells, but express highly in the nucleus. s- lap/ GFP融合蛋白可在人 Hela細胞中表達 ,并主要定位于細胞核。
- Flow cytometry (FCM) was used to examine the effects of 4-HPR with or without DDP on cell cycle and apoptosis of HeLa cells. 采用流式細胞儀測定4-HPR、DDP、4-HPR+DDP對HeLa腫瘤細胞系細胞增殖周期和細胞凋亡的變化。
- The CPE inhibition assay was used to test the antiviral activity of GD4 on RSV in Hela cells. 采用細胞病變抑制實(shí)驗,觀(guān)察GD4在Hela細胞中對RSV的抑制作用。
- G418-resistant clones of Hela cells were obtained and confirmed as positive by RT-PCR. 重組質(zhì)粒轉染Hela細胞后,經(jīng)G418篩選成功獲得陽(yáng)性克隆。
- Aim To evaluate the manner and molecular mechanism of HeLa cell death induced by Coxsackie virus B3(CVB3). 目的評價(jià)柯薩奇病毒B3CVB3致HeLa細胞死亡的方式及分子機制。
- Gpd-Tp92 fusion gene constructed in pcDNA3.1 (+) vector could express a fusion protein in Hela cells. pcDNA3.;1(+)/Gpd-Tp92在HeLa細胞中能有效表達。
- Both genes were cloned into GFP expression vector pIRES2 EGFP and transfected into HeLa cells. 將上述基因克隆入綠色熒光蛋白(GFP)表達載體pIRES2-EGFP中,轉染人HeLa細胞,在熒光顯微鏡和電鏡下觀(guān)察轉染細胞的形態(tài)和結構。
- The KGF and KGFR genes were expressed in the Hela cells. The KGF and KGFR proteins were expressed in the Hela cells. 從基因水平和蛋白水平證實(shí)KGF及KGFR在Hela細胞中有表達。
- The augment of G 1 ratio in the proliferation cycle of Hela cells presents a concentration-dependent manner. 細胞周期分析顯示槲皮素組 G 1 期細胞比例明顯增多，呈量-效關(guān)系。
- Role of Mitochondria in HeLa Cell Apoptosis Induced by Hematoporphyrin monomethyl ether- PDT[J]. 引用該論文 丁新民;劉凡光;顧瑛;曾晶;戴維德;李曉松.
- Stably transfected and control Hela cell lines were injected subcutaneously into female Balb/c/nu mice. 穩定轉染Ang-2的細胞注射Balb/c裸鼠皮下。
