After renal ischemia reperfusion, the high expression of iNOSmRNA in tubules is an important molecular mechanism of renal ischemia reperfusion injury (2) Keep cNOS/iNOS constant may be important for mudulation of renal blood flow and GFR.
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- 缺血再灌注后 ,皮質(zhì)腎小管上皮中iNOSmRNA的高表達是導致腎缺血再灌注損傷的重要分子機制。 (2 )cNOS/iN OS比值與缺血再灌注過(guò)程中腎功能的變化密切相關(guān) ,該比值的恒定對腎血流量和腎小球濾過(guò)率(GFR)的調節可能具有重要意義
